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LOW BACK PAIN
1980-
2009
Second most commen symptom
Affect all age groups / all economic groups
Globally 54% increase in the last 25 yrs
12 – 33% of adult population have low back pain
LOW BACK PAIN
History
Natural history of low back pain
 80%-90% resolves in 1 months how back pain develops
 20%-30% remains chronic
 5%-10% disabling Muscle imbalances
dysfunction
Condutions -
herniated disc
Pain
Successful
treatment
needs to
begin here
Most
treatments
fail
obesity, smoking,
gender
lifting, vibration,
prolonged sitting
job
dissatisfaction
low back pain
Cause of lower back pain
Mechanical 80%
- Lumbar sprain , strain
- Facet joint arthritis
- proplased intervertebral disc
- spondylosis
- Disc anular tears
Infection
- Osteomyelitis
- Discitis
Traumatic
- Spondylolysis
- spondylolisthesis
- Sprain, stain
- spinal/ foraminal stenosis
- vertebral fracture
Metabolic
- osteoporosis
- osteomalacia
- Paget disease
• Viscerogenic ( refered)
- Genitourinary disease
-Gynaecological disease
• Vasculogenic
- Aortic aneurysem
• Psychogen
-Somatization disorder
- Myofascial pain
- Fibromyalgia
Mechanical low back pain
No clearly defined pathology
No significant clinical or radiological findings
Diagnosis of exclusion
• Aggrevated with movement and relited by rest
• Patients may complain of symptoms on standing up from supine or
seated positions , and pain on turning over bed .
Clinical features
• dull pain
• Aggrevated by
- physicial activity
- stooping
- lifting
- turning and coughing
• No neurological signs and symptoms
• it is not possible to clinically distinguish the source of pain between disc,
facet joint , muscle , ligaments and SI joint .
• Pain on flexion may be related to discogenic pain and facetogenic pain
may be aggrevated by hyperextension.
Features of pain
• Location : upper , middle or lower back. Disck prolaps and degenerative
spondylitis occur in the lower lumbar spine, infection and trauma occur in
the dorso lumbar spine.
• Onset : it has a history of trauma or disc prolapse. Pain resulting from a
routine activity such as twisting to pull something out of a drawer.
• Location of pain: pain arising from tend or musle injury is localized,
whereas that originating from deeper structure is diffuse. Pain referred to a
dermatome of the lower limb with neurological signs menas nerve root
entrapment.
• Progres of pain: in traumatic condition and acut disc prolaps pain is
maximum at the onset, and then gradually subside over days or weeks.
Back pain to disc prolaps has remission and exacerbation. Arthrotic and
spondylotic pain are more constant and aggrevated by activity. Pain to
infection and tumors take progressive cours ,with nothing causig relief.
• Releiving and aggravitating factors: most back pains are worsended by
activity and relieved by rest. Pain due to ankylosing spondylitis and
seronegative arthritis SSA is typically worse after rest and improves with
activity. Sever back pain at night ant relieved by aspirin indicate to bening
tumor. Pain initiated on walking or standing and relieved by rest is a
feature of spial stenosis. An increase of pain due menstruaction may
indicate a gynaecological pathology.
Physical examination
• Position- inspection
palpation
SLRT
Straight Leg Test Variant
Femoral Stretch Test (L2-4)
• L4 Motor Exam
• To test L4 strength, have the patient
slightly bend the knee and kick out
as you keep pressure against the leg.
Be sure to compare both sides to
see if one side has weakness relative
to the other.
L5 Motor Exam
• L5 Motor Exam
• To test L5 strength, hold pressure over the large
toes and ask the patient to dorsiflex the big toes
and foot towards up. Compare both sides for
relative weakness.
Test for L5 weakness with walking on heels in normal patient.
If one foot is unable to lift toes off ground, could suggest L5
weakness on that side.
Test for S1 weakness with walking on toes in
normal patient. If one foot is unable to lift
heal off ground, could suggest S1 weakness
on that side.
S 1 Motor exam
Sensory Exam
• In the sensory exam, again
focusing on L4, L5 & S1, we will
look at specific dermatomal
regions as noted in the image.
If possible, use a
monofilament. If not present,
you can use your fingers or the
tip of a tongue depressor to
test for sensation.
L4 Sensory Exam
Focus on the anterior/lateral aspect of the thigh.
L5 Sensory Exam
S1- sensory exam
Reflex Exam
• L4 is tested by the patellar reflex.
L5 Reflex Exam
L5 is tested by the medial hamstring reflex.
S1 Reflex Exam
Investigation
• X ray – spondylolisthesis and retrolisthesis AP. Lateral , oblique
• CT And MRI- disc degeneration- Modic , anular teat, facet
arthropaty
• Blood tests - WBC, ESR, CRP infection and inflammatory,
Neoplastic
• Bone scan –facet joint uptake, SI, Metastatic neo, Vertebral fr.
Sydney
19 mil
consulti
ons
45 study
1995-2017 4.3 mil
Imaging
1: 4 primary care 1:3
emergency
When to consider imaging?
• Neurological deficit
• Significant of trauma
• Signs of infection
• Ca
• Osteoporosis
• Age >50
• Congenital spine problems
• History of a previous spine injury or back surgery
• History of long term steroid use or drug abuse
• Back pain > 4 wks
• Several episodes of severe pain.
Treatment
Rest
Analgesic
Physiotherapy
Education
Surgery
50 Obese-
gastroplasty
50 non-
obese
another
OP
LBP 29
obese
pre OP
58%
After OP- 10
patient LWP,-
reduce dose
of medication
19 no LBP
12 (24%),
LWB pre
OP.
Management of low back pain
• Surgery
1. desctectomy
2. decompresion ( laminectomy )
3. spinal stabilization
4. verteboplasty / Kyphoplasty
5. Foraminotomy
6. Nuncleoplasty
7. Artificial disc replacement
• Implanted nerve stimulator
1. Spinal cord stimulation
2. dorsal root ganglion stimulation
3. peripheral nerve stimulation
Who needs Surgery
unstabile spine
acute fracture with neurological deficit
severe stenosis
after failure of aggressive non operative treatmet
Tu
progessive neurological deficite
sy Cauda equinea
Lumbar Disc Herniation is
a very common cause of
low back pain and
radicular leg pain, most
commonly affecting the
L4-L5 and L5-S1 levels.
Diagnosis is made with
MRI studies of the
lumbar spine.
Treatment is a trial of
nonoperative
management with
NSAIDs and physical
therapy. Surgical
laminotomy and
discectomy is indicated
for progressive disabling
pain that has failed
nonoperative
management, progressive
neurological deficits, or
cauda equina syndrome.
Epidemiology
peak
incidence is
4th and 5th
decades
lifetime
prevalence
of 10%
only ~5%
become
symptomatic
3:1
male:female
ratio
recurrent torsional strain leads to tears of outer annulus which leads to
herniation of nucleus pulposis
location
L5/S1 most common level
95% involve L4/5 or L5/S1 levels
• Annulus fibrous
- typ I collagen , water and proteoglycan
- characterized by extensibility and tensile strength
high collagen / low proteoglycan ratio (low % dry
weight of
proteoglycans)
• nucleus pulposus
- composed of type II collagen, water, and proteoglycans
- characterized by compressibility
- low collagen / high proteoglycan ratio (high % dry
weight of proteoglycans)
- proteoglycans interact with water and resist
compression
- a hydrated gel due to high polysacharide content and
high water content (88%)
Classification
Central : cauda equinea Sy CSE
• back pain(mostcommon)
• unilateralor bilateralleg pain(2ndmost
• common)
• saddleanesthesia
• bladderdysfunction
• unilateralor bilateralsensory changesin
legs
• unilateral orbilateralmotor weaknessin
• legs
• sexual dysfunction (impotencein men)
• bowel dysfunction
posterolateral (paracentral)
most common (90-95%)
PLL is weakest here
affects the traversing/descending/lower
nerve root
at L4/5 affects L5 nerve root
classification
Foraminal (far lateral, extraforaminal)
less common (5-10%)
affects exiting/upper nerve root
at L4/5 affects L4 nerve root
herniated disc material directly compresses dorsal root
ganglion
can manifest with more severe pain than traditional
posterolateral
disc herniation
Stage of disc herniation
RADICULOPATHY
Radiculopathy typically present with symptoms of radicular pain.
the absolute diagnose of radiculopathy is cinclusively made by
electrodiagnostic studies ( EMG/ NCV)
Postive neurological finding also make e strong working diagnosis.
Postive neurological findings include
•Deep tendon reflex change
•Motor weakness
•Sensory change
•Should NOT be use to describe lower extremity pain.
•It is a diagnois , pathology .
RADICULAR PAIN
• Lumbar Spinal Stenosis is a degenerative spinal condition
characterized by the narrowing of the lumbar spinal canal due
to a variety of bony or soft tissues structures.
• indence
most common reason for lumbar spine surgery in patients > 65
years
old
seen in 20-25%
Classification
• Acquired
• degenerative/spondylotic changes
(most common)
• post-surgical
• post-traumatic (vertebral fractures)
• inflammatory (ankylosing
spondylitis)
• secondary to systemic diseases
(Paget disease, acromegaly,
fluorosis)
• short pedicles with medially placed
facets
• can be subdivided into
• idiopathic
• developmental (achondroplasia)
Central stenosis < 10 mm AP axial CT/ MRI
Paracentral stenosis( lateral recesus )- descending nerve root compresion
foraminal stenosis - exiting nereve root compression
Extraforaminal stenosis- exiting nerve root compresion
• back pain
referred buttock pain
• leg pain
often unilateral
• neurogenic claudication
pain worse with extension (walking, standing upright)
pain relieved with flexion (sitting, leaning over shopping cart, sleeping in fetal
position)
• weakness
• bladder disturbances
recurrent UTI present in up to 10% due to autonomic sphincter dysfunction
• cauda equina syndrome (rare)
Spondylolisthesis
Spondylolisthesis
is the displacement
of one spinal
vertebra compared
to another
Cause
Degenerative
Traumatic
Dysplastic
Isthmic
Pathologic
Post-surgical/iatrogenic
• mechanical/ back pain
• most common presenting
symptom
• usually relieved with rest
and sitting
• defined as buttock and leg
pain/discomfort caused by upright
walking
• relieved by sitting
• not relieved by standing in one
place (as is vascular claudication)
• may be unilateral or bilateral
Classification-
LOW BACK PAIN. Dr Haki Selaj Residency in Kosovo QKUK
LOW BACK PAIN. Dr Haki Selaj Residency in Kosovo QKUK
LOW BACK PAIN. Dr Haki Selaj Residency in Kosovo QKUK
LOW BACK PAIN. Dr Haki Selaj Residency in Kosovo QKUK

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LOW BACK PAIN. Dr Haki Selaj Residency in Kosovo QKUK

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  • 6. Second most commen symptom Affect all age groups / all economic groups Globally 54% increase in the last 25 yrs 12 – 33% of adult population have low back pain
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  • 11. Natural history of low back pain  80%-90% resolves in 1 months how back pain develops  20%-30% remains chronic  5%-10% disabling Muscle imbalances dysfunction Condutions - herniated disc Pain Successful treatment needs to begin here Most treatments fail
  • 14. Cause of lower back pain
  • 15. Mechanical 80% - Lumbar sprain , strain - Facet joint arthritis - proplased intervertebral disc - spondylosis - Disc anular tears Infection - Osteomyelitis - Discitis Traumatic - Spondylolysis - spondylolisthesis - Sprain, stain - spinal/ foraminal stenosis - vertebral fracture Metabolic - osteoporosis - osteomalacia - Paget disease
  • 16. • Viscerogenic ( refered) - Genitourinary disease -Gynaecological disease • Vasculogenic - Aortic aneurysem • Psychogen -Somatization disorder - Myofascial pain - Fibromyalgia
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  • 19. No clearly defined pathology No significant clinical or radiological findings Diagnosis of exclusion
  • 20. • Aggrevated with movement and relited by rest • Patients may complain of symptoms on standing up from supine or seated positions , and pain on turning over bed .
  • 21. Clinical features • dull pain • Aggrevated by - physicial activity - stooping - lifting - turning and coughing • No neurological signs and symptoms • it is not possible to clinically distinguish the source of pain between disc, facet joint , muscle , ligaments and SI joint . • Pain on flexion may be related to discogenic pain and facetogenic pain may be aggrevated by hyperextension.
  • 22. Features of pain • Location : upper , middle or lower back. Disck prolaps and degenerative spondylitis occur in the lower lumbar spine, infection and trauma occur in the dorso lumbar spine. • Onset : it has a history of trauma or disc prolapse. Pain resulting from a routine activity such as twisting to pull something out of a drawer. • Location of pain: pain arising from tend or musle injury is localized, whereas that originating from deeper structure is diffuse. Pain referred to a dermatome of the lower limb with neurological signs menas nerve root entrapment.
  • 23. • Progres of pain: in traumatic condition and acut disc prolaps pain is maximum at the onset, and then gradually subside over days or weeks. Back pain to disc prolaps has remission and exacerbation. Arthrotic and spondylotic pain are more constant and aggrevated by activity. Pain to infection and tumors take progressive cours ,with nothing causig relief. • Releiving and aggravitating factors: most back pains are worsended by activity and relieved by rest. Pain due to ankylosing spondylitis and seronegative arthritis SSA is typically worse after rest and improves with activity. Sever back pain at night ant relieved by aspirin indicate to bening tumor. Pain initiated on walking or standing and relieved by rest is a feature of spial stenosis. An increase of pain due menstruaction may indicate a gynaecological pathology.
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  • 32. SLRT
  • 33. Straight Leg Test Variant
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  • 37. • L4 Motor Exam • To test L4 strength, have the patient slightly bend the knee and kick out as you keep pressure against the leg. Be sure to compare both sides to see if one side has weakness relative to the other.
  • 38. L5 Motor Exam • L5 Motor Exam • To test L5 strength, hold pressure over the large toes and ask the patient to dorsiflex the big toes and foot towards up. Compare both sides for relative weakness.
  • 39. Test for L5 weakness with walking on heels in normal patient. If one foot is unable to lift toes off ground, could suggest L5 weakness on that side.
  • 40. Test for S1 weakness with walking on toes in normal patient. If one foot is unable to lift heal off ground, could suggest S1 weakness on that side.
  • 41. S 1 Motor exam
  • 42. Sensory Exam • In the sensory exam, again focusing on L4, L5 & S1, we will look at specific dermatomal regions as noted in the image. If possible, use a monofilament. If not present, you can use your fingers or the tip of a tongue depressor to test for sensation.
  • 43. L4 Sensory Exam Focus on the anterior/lateral aspect of the thigh.
  • 46. Reflex Exam • L4 is tested by the patellar reflex.
  • 47. L5 Reflex Exam L5 is tested by the medial hamstring reflex.
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  • 50. Investigation • X ray – spondylolisthesis and retrolisthesis AP. Lateral , oblique • CT And MRI- disc degeneration- Modic , anular teat, facet arthropaty • Blood tests - WBC, ESR, CRP infection and inflammatory, Neoplastic • Bone scan –facet joint uptake, SI, Metastatic neo, Vertebral fr.
  • 51. Sydney 19 mil consulti ons 45 study 1995-2017 4.3 mil Imaging 1: 4 primary care 1:3 emergency
  • 52. When to consider imaging? • Neurological deficit • Significant of trauma • Signs of infection • Ca • Osteoporosis • Age >50 • Congenital spine problems • History of a previous spine injury or back surgery • History of long term steroid use or drug abuse • Back pain > 4 wks • Several episodes of severe pain.
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  • 58. 50 Obese- gastroplasty 50 non- obese another OP LBP 29 obese pre OP 58% After OP- 10 patient LWP,- reduce dose of medication 19 no LBP 12 (24%), LWB pre OP.
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  • 66. Management of low back pain • Surgery 1. desctectomy 2. decompresion ( laminectomy ) 3. spinal stabilization 4. verteboplasty / Kyphoplasty 5. Foraminotomy 6. Nuncleoplasty 7. Artificial disc replacement • Implanted nerve stimulator 1. Spinal cord stimulation 2. dorsal root ganglion stimulation 3. peripheral nerve stimulation
  • 67. Who needs Surgery unstabile spine acute fracture with neurological deficit severe stenosis after failure of aggressive non operative treatmet Tu progessive neurological deficite sy Cauda equinea
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  • 71. Lumbar Disc Herniation is a very common cause of low back pain and radicular leg pain, most commonly affecting the L4-L5 and L5-S1 levels. Diagnosis is made with MRI studies of the lumbar spine. Treatment is a trial of nonoperative management with NSAIDs and physical therapy. Surgical laminotomy and discectomy is indicated for progressive disabling pain that has failed nonoperative management, progressive neurological deficits, or cauda equina syndrome.
  • 72. Epidemiology peak incidence is 4th and 5th decades lifetime prevalence of 10% only ~5% become symptomatic 3:1 male:female ratio
  • 73. recurrent torsional strain leads to tears of outer annulus which leads to herniation of nucleus pulposis location L5/S1 most common level 95% involve L4/5 or L5/S1 levels
  • 74. • Annulus fibrous - typ I collagen , water and proteoglycan - characterized by extensibility and tensile strength high collagen / low proteoglycan ratio (low % dry weight of proteoglycans) • nucleus pulposus - composed of type II collagen, water, and proteoglycans - characterized by compressibility - low collagen / high proteoglycan ratio (high % dry weight of proteoglycans) - proteoglycans interact with water and resist compression - a hydrated gel due to high polysacharide content and high water content (88%)
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  • 76. Classification Central : cauda equinea Sy CSE • back pain(mostcommon) • unilateralor bilateralleg pain(2ndmost • common) • saddleanesthesia • bladderdysfunction • unilateralor bilateralsensory changesin legs • unilateral orbilateralmotor weaknessin • legs • sexual dysfunction (impotencein men) • bowel dysfunction
  • 77. posterolateral (paracentral) most common (90-95%) PLL is weakest here affects the traversing/descending/lower nerve root at L4/5 affects L5 nerve root
  • 78. classification Foraminal (far lateral, extraforaminal) less common (5-10%) affects exiting/upper nerve root at L4/5 affects L4 nerve root herniated disc material directly compresses dorsal root ganglion can manifest with more severe pain than traditional posterolateral disc herniation
  • 79. Stage of disc herniation
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  • 81. RADICULOPATHY Radiculopathy typically present with symptoms of radicular pain. the absolute diagnose of radiculopathy is cinclusively made by electrodiagnostic studies ( EMG/ NCV) Postive neurological finding also make e strong working diagnosis. Postive neurological findings include •Deep tendon reflex change •Motor weakness •Sensory change •Should NOT be use to describe lower extremity pain. •It is a diagnois , pathology . RADICULAR PAIN
  • 82. • Lumbar Spinal Stenosis is a degenerative spinal condition characterized by the narrowing of the lumbar spinal canal due to a variety of bony or soft tissues structures. • indence most common reason for lumbar spine surgery in patients > 65 years old seen in 20-25%
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  • 84. Classification • Acquired • degenerative/spondylotic changes (most common) • post-surgical • post-traumatic (vertebral fractures) • inflammatory (ankylosing spondylitis) • secondary to systemic diseases (Paget disease, acromegaly, fluorosis) • short pedicles with medially placed facets • can be subdivided into • idiopathic • developmental (achondroplasia)
  • 85. Central stenosis < 10 mm AP axial CT/ MRI Paracentral stenosis( lateral recesus )- descending nerve root compresion foraminal stenosis - exiting nereve root compression Extraforaminal stenosis- exiting nerve root compresion
  • 86. • back pain referred buttock pain • leg pain often unilateral • neurogenic claudication pain worse with extension (walking, standing upright) pain relieved with flexion (sitting, leaning over shopping cart, sleeping in fetal position) • weakness • bladder disturbances recurrent UTI present in up to 10% due to autonomic sphincter dysfunction • cauda equina syndrome (rare)
  • 87.
  • 88. Spondylolisthesis Spondylolisthesis is the displacement of one spinal vertebra compared to another
  • 90. • mechanical/ back pain • most common presenting symptom • usually relieved with rest and sitting • defined as buttock and leg pain/discomfort caused by upright walking • relieved by sitting • not relieved by standing in one place (as is vascular claudication) • may be unilateral or bilateral